Miniturization of Scintillation Proximity Assays to 384 Well Microplate Format

A HIGH THROUGHPUT SCREENING ASSAY FOR THIAMINE PYROPHOSPHATE KINASE ACTIVITY USING SPA
1
Greet Vanhoof, 2*Jenny Berry, 2Mark Harvey, 2Molly Price-Jones and 2Kelvin Hughes.
1
Janssen Research Foundation, Beerse, Belgium.
Amersham Biosciences UK Limited, Amersham Place, Little Chalfont, Buckinghamshire, England, HP7 9NA (Telephone +44 (0) 29 2052 6417,
Fax +44 (0) 29 2052 6474, e: mail [email protected])
2
-3
ne
ly
G
M
m
0.25
0.50
0.75
1.00
2.0
KM = 0.04mM
1.0
0.0
0.1
0.2
0.3
0.4
0.5
0.6
0.7
0.8
1.0
1.5
2.0
2.5
SPA bead (mg/well)
Figure 3. Effect of increasing concentrations of YSi
bead on SPA assay signal. Assays contained 1.0µg
enzyme/well. Values are means ±SEM (n=2).
As can be seen from Figure 2, both the type of buffer,
and the buffer concentration, appeared to have a
considerable effect on the SPA cpm obtained. The use
of glycine buffer resulted in ~40% increase in SPA cpm
compared to tris buffer. Increasing the glycine buffer
concentration from 5 to 20mM appeared to reduce the
SPA cpm by ~50%.
A titration of SPA bead (Fig.3) indicated that the
maximum signal: background was obtained with 1mg
YSi bead/well.
Experiments indicated that the assay signal was linear
up
to
1.25µg/well
added
enzyme
(Fig.4).
10
100
1000
pyrithiamine concentration (µ
µ M)
Figure 6. Inhibition of TPPK by pyrithiamine. Values
are means ±SEM (n=3).
included 8 wells of positive controls (in the presence of
TPPK enzyme) and 8 wells of negative controls (in the
absence of TPPK), testing a total of 3360 compounds.
The average of the cpm values for the positive controls
was 6245 cpm with a CV of 7%. The average cpm
value for the negative control was 816 cpm with a CV
of 8%. To evaluate the quality of the HTS we
calculated the Z’ value(7) and found it was 0.7. The
quality of the assay allows implementation in an HTS
system with delivery of precise and reproducible data.
0.9
[3H]thiamine concentration (mM)
0.5
0.5
3.0
(-) enzyme
20.0
IC50 = 19.4µ
µM
1
-3
20
-3
(+) enzyme
40.0
1.0
1.25
Figure 4. Effect of increasing enzyme concentration.
Values are means ±SEM (n=3).
SPA cpm (x10 )
M
5m
-3
5.0
Enzyme concentration (µ
µ g/well)
Figure 2. Effect of buffer type and concentration on
SPA assay signal. Both tris and glycine buffers
(containing 13µM ATP and 2mM MnSO4) were used at
pH 8.6. Values are means ±SEM (n=2).
SPA cpm (x10 )
10
0.00
buffer
0.0
Method and Results
SPA assays contained TPPK (0.5µg), unlabelled
3
thiamine (10nmol) and [ H]thiamine (70pmol, 1µCi) in a
total volume of 100µl 5mM glycine buffer, pH 8.6,
containing 13µM ATP and 2mM MnSO4. Assays were
incubated at room temperature for 20 minutes before
being stopped by the addition of 50µl de-ionized water
containing 1mg YSi SPA beads. Non-specific binding
was determined in the absence of TPPK.
Assays were counted for 1 minute/well using a
TopCount microplate scintillation counter. Library
compounds were added in DMSO to give a final
concentration of 1% DMSO (v/v) and 10-5M test
compound in the assay well.
1.5
15
ci
ne
ci
ly
G
M
m
20
5m
M
Tr
Tr
is
is
5.0
SPA cpm (x10 )
-3
(-) enzyme
10.0
60.0
Figure 1. Diagrammatic representation of the TPPK
SPA assay.
20
(+) enzyme
SPA cpm (x10 )
15.0
SPA cpm (x10 )
Introduction
Thiamine pyrophosphate kinase (TPPK: E.C. 2.7.6.2)
converts thiamine to thiamine pyrophosphate (TPP)
which acts as the major metabolic thiamine signal
within the cell (1).
TPP functions as a cofactor for many enzymes involved
in
carbohydrate
metabolism,
and
in
Schizosaccharomyces pombe, has been shown to be a
critical metabolic signal for mating. TTP is also thought
to be important in phosphate metabolism and growth in
yeast (2). Therefore, inhibition of TTPK may be a target
for anti-fungal therapy.
Current methods for analysing TTPK activity such as
HPLC (3) and ion exchange chromatography(4) are not
amenable to high throughput screening; therefore, we
have developed a scintillation proximity assay (SPA) to
measure TPPK activity.
In this assay, TPPK from Saccharomyces cerevisiae
was used to convert [3H]thiamine to [3H]TPP which
was selectively captured using yttrium silicate (YSi)
SPA beads. (Fig. 1).
Figure 5. Michealis-Menten plot for TPPK with
[3H]thiamine as substrate. Values are means ±SEM
(n=3).
Non-linear regression analysis of the substrate
concentration curve obtained with [3H]thiamine (Fig.5)
gave a KM value of 40µM (± 8µM). This is in good
agreement with the previously reported KM values for
procaryotic TPPK (75µM for Saccharomyces cerevisiae
TPPK(5), 6µM for Saccharomyces pombe TPPK(2) and
38µM for Paracoccus denitrificans TPPK (6)).
Pyrithiamine, a substrate analogue and thiamine
antagonist, is thought to be a direct competitive
inhibitor of TPPK. Reported Ki values for pyrithiamine of
the procaryotic TPPK are 6µM(2) and 19µM(6). As can
be seen from Fig.6, inhibition studies indicate that
pyrithiamine does inhibit Saccharomyces cerevisiae
TPPK with an estimated IC50 value of 19µM.
The robustness of this SPA assay was tested using a
high throughput screening system, screening 42 x 96well plates of our compound library, where each plate
.
Conclusions
•
•
We have developed a high throughput
screening assay to measure the
inhibition of TPPK activity.
The assay is robust, and can be used
for automated screening of a large
number of compounds for discovery of
hits and development of therapeutic
leads.
References
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